атипичные формы генитального герпеса клиника и лечение

At children defeat of a smooth leather{skin} on stops is characterized мелкопластинчатым by a peeling on an internal surface of trailer phalanxes of fingers, is $more often III and IV or there are superficial, less often deep cracks, mainly in III and IV intermanual складках or under fingers, гиперемией and мацерацией. On soles the leather{skin} can be not changed or amplified{strengthened} skin figure, is sometimes observed кольцевидное a peeling. Subjectively patients are disturbed with an itch. At children is $more often, than at adults, arise экссудативные forms of defeat with formation{education} пузырьков, becoming wet, экземоподобных the centers. They appear not only on stops, but also brushes. Подробнее...

By special works [6] it is established, that the genetic control over synthesis of the general{common} IgE is the paramount factor in development the HELL. It is shown, that main рецессивный a locus, детерминирующий a high level сывороточного IgE, influences development of various symptoms атопических diseases. In cases when the patient suffers only an asthma or only the HELL, the effect of influence of the main gene is similar with рецессивным inheritance. If joins поллиноз, most likely, the question is about кодоминантном inheritance. It is shown, that connection to a clinical picture of additional symptoms атопии testifies to presence of prepotent influence [5]. Last years communication{connection} атопических diseases with the certain antigenes of the main complex гистосовместимости is found out, the positive association атопического дерматита with antigenes HLA A24,-В5,-В9,-В12 and-В27 in particular is established. It is established, that the fact of reaction of immune system on an antigene is impossible without system of the main complex гистосовместимости as cooperation of cells{cages} in the immune answer is provided at participation of antigenes of system HLA [7,8]. Last data testify to participation in патогенезе the HELL of mechanisms of genetic regulation of the immune answer connected to a HLA-marker [9]. Подробнее...